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BR Harris

First name:
BR
Last name:
Harris
Mulholland, P. J., Stepanyan, T. D., Self, R. L., Hensley, A. K., Harris, B. R., Kowalski, A., et al. Corticosterone and dexamethasone potentiate cytotoxicity associated with oxygen-glucose deprivation in organotypic cerebellar slice cultures. Neuroscience, 136(1), 259-67. https://doi.org/10.1016/j.neuroscience.2005.07.043 (Original work published 2005)
Mulholland, P. J., Self, R. L., Harris, B. R., Little, H. J., Littleton, J. M., & Prendergast, M. A. (2005). Corticosterone increases damage and cytosolic calcium accumulation associated with ethanol withdrawal in rat hippocampal slice cultures. Alcoholism, Clinical And Experimental Research, 29(5), 871-81. https://doi.org/10.1097/01.alc.0000163509.27577.da
Self, R. L., Mulholland, P. J., Harris, B. R., Nath, A., & Prendergast, M. A. (2004). Cytotoxic effects of exposure to the human immunodeficiency virus type 1 protein Tat in the hippocampus are enhanced by prior ethanol treatment. Alcoholism, Clinical And Experimental Research, 28(12), 1916-24. https://doi.org/10.1097/01.alc.0000148108.93782.05
Mulholland, P. J., Self, R. L., Harris, B. R., Littleton, J. M., & Prendergast, M. A. (2004). Choline exposure reduces potentiation of N-methyl-D-aspartate toxicity by corticosterone in the developing hippocampus. Brain Research. Developmental Brain Research, 153(2), 203-11. https://doi.org/10.1016/j.devbrainres.2004.08.008 (Original work published 2004)
Mulholland, P. J., Self, R. L., Harris, B. R., Littleton, J. M., & Prendergast, M. A. (-)-nicotine ameliorates corticosterone's potentiation of N-methyl-d-aspartate receptor-mediated cornu ammonis 1 toxicity. Neuroscience, 125(3), 671-82. https://doi.org/10.1016/j.neuroscience.2004.02.007 (Original work published 2004)
Prendergast, M. A., Harris, B. R., Mullholland, P. J., JA, 2nd B., Gibson, D. A., Holley, R. C., & Littleton, J. M. Hippocampal CA1 region neurodegeneration produced by ethanol withdrawal requires activation of intrinsic polysynaptic hippocampal pathways and function of N-methyl-D-aspartate receptors. Neuroscience, 124(4), 869-77. https://doi.org/10.1016/j.neuroscience.2003.12.013 (Original work published 2004)
Self, R. L., Mulholland, P. J., Nath, A., Harris, B. R., & Prendergast, M. A. (2004). The human immunodeficiency virus type-1 transcription factor Tat produces elevations in intracellular Ca2+ that require function of an N-methyl-D-aspartate receptor polyamine-sensitive site. Brain Research, 995(1), 39-45. https://doi.org/10.1016/j.brainres.2003.09.052 (Original work published 2004)
Harris, B. R., Gibson, D. A., Prendergast, M. A., Blanchard, J. A., Holley, R. C., Scotland, R. L., et al. (2003). The neurotoxicity induced by ethanol withdrawal in mature organotypic hippocampal slices might involve cross-talk between metabotropic glutamate type 5 receptors and N-methyl-D-aspartate receptors. Alcoholism, Clinical And Experimental Research, 27(11), 1724-35. https://doi.org/10.1097/01.ALC.0000093601.33119.E3
Mulholland, P. J., Harris, B. R., Wilkins, L. H., Self, R. L., Blanchard, J. A., Holley, R. C., et al. (1969). Opposing effects of ethanol and nicotine on hippocampal calbindin-D28k expression. Alcohol (Fayetteville, N.y.), 31(1-2), 1-10. https://doi.org/10.1016/j.alcohol.2003.09.001 (Original work published 1969)
Gibson, D. A., Harris, B. R., Prendergast, M. A., JA, 2nd B., Holley, R. C., Pedigo, N. W., et al. (2003). Polyamines contribute to ethanol withdrawal-induced neurotoxicity in rat hippocampal slice cultures through interactions with the NMDA receptor. Alcoholism, Clinical And Experimental Research, 27(7), 1099-106. https://doi.org/10.1097/01.ALC.0000075824.10502.DD