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PJ Mulholland

First name:
PJ
Last name:
Mulholland
Self, R. L., Mulholland, P. J., Harris, B. R., Nath, A., & Prendergast, M. A. (2004). Cytotoxic effects of exposure to the human immunodeficiency virus type 1 protein Tat in the hippocampus are enhanced by prior ethanol treatment. Alcoholism, Clinical And Experimental Research, 28(12), 1916-24. https://doi.org/10.1097/01.alc.0000148108.93782.05
Mulholland, P. J., Teppen, T. L., Miller, K. M., Sexton, H. G., Pandey, S. C., & Swartzwelder, H. S. (2018). Donepezil reverses dendritic spine morphology adaptations and Fmr1 epigenetic modifications in hippocampus of adult rats after adolescent alcohol exposure. Alcoholism, Clinical And Experimental Research. https://doi.org/10.1111/acer.13599 (Original work published 2018)
Mulholland, P. J., & Prendergast, M. A. (2002). Post-insult exposure to (+/-) kavain potentiates N-methyl-D-aspartate toxicity in the developing hippocampus. Brain Research, 945(1), 106-13. https://doi.org/10.1016/s0006-8993(02)02745-2 (Original work published 2002)
Prendergast, M. A., Rogers, D. T., Mulholland, P. J., Littleton, J. M., LH, W., Self, R. L., & Nath, A. (2002). Neurotoxic effects of the human immunodeficiency virus type-1 transcription factor Tat require function of a polyamine sensitive-site on the N-methyl-D-aspartate receptor. Brain Research, 954(2), 300-7. https://doi.org/10.1016/s0006-8993(02)03360-7 (Original work published 2002)
Mulholland, P. J., & Prendergast, M. A. (2003). Transection of intrinsic polysynaptic pathways reduces N-methyl-D-aspartate neurotoxicity in hippocampal slice cultures. Neuroscience Research, 46(3), 369-76. https://doi.org/10.1016/s0168-0102(03)00102-0
Mulholland, P. J., Harris, B. R., Wilkins, L. H., Self, R. L., Blanchard, J. A., Holley, R. C., et al. (1969). Opposing effects of ethanol and nicotine on hippocampal calbindin-D28k expression. Alcohol (Fayetteville, N.y.), 31(1-2), 1-10. https://doi.org/10.1016/j.alcohol.2003.09.001 (Original work published 1969)
Self, R. L., Mulholland, P. J., Nath, A., Harris, B. R., & Prendergast, M. A. (2004). The human immunodeficiency virus type-1 transcription factor Tat produces elevations in intracellular Ca2+ that require function of an N-methyl-D-aspartate receptor polyamine-sensitive site. Brain Research, 995(1), 39-45. https://doi.org/10.1016/j.brainres.2003.09.052 (Original work published 2004)
Mulholland, P. J., Self, R. L., Harris, B. R., Littleton, J. M., & Prendergast, M. A. (-)-nicotine ameliorates corticosterone's potentiation of N-methyl-d-aspartate receptor-mediated cornu ammonis 1 toxicity. Neuroscience, 125(3), 671-82. https://doi.org/10.1016/j.neuroscience.2004.02.007 (Original work published 2004)
Mulholland, P. J., Self, R. L., Harris, B. R., Littleton, J. M., & Prendergast, M. A. (2004). Choline exposure reduces potentiation of N-methyl-D-aspartate toxicity by corticosterone in the developing hippocampus. Brain Research. Developmental Brain Research, 153(2), 203-11. https://doi.org/10.1016/j.devbrainres.2004.08.008 (Original work published 2004)
Butler, T. R., Self, R. L., Smith, K. J., Sharrett-Field, L. J., Berry, J. N., Littleton, J. M., et al. (2010). Selective vulnerability of hippocampal cornu ammonis 1 pyramidal cells to excitotoxic insult is associated with the expression of polyamine-sensitive N-methyl-D-asparate-type glutamate receptors. Neuroscience, 165(2), 525-34. https://doi.org/10.1016/j.neuroscience.2009.10.018 (Original work published 2010)