Self, R., Mulholland, P., Harris, B., Nath, A., & Prendergast, M. (2004). Cytotoxic effects of exposure to the human immunodeficiency virus type 1 protein Tat in the hippocampus are enhanced by prior ethanol treatment. Alcoholism, Clinical and Experimental Research, 28(12), 1916-24. https://doi.org/10.1097/01.alc.0000148108.93782.05

Mulholland, P., Teppen, T., Miller, K., Sexton, H., Pandey, S., & Swartzwelder, H. (2018). Donepezil reverses dendritic spine morphology adaptations and Fmr1 epigenetic modifications in hippocampus of adult rats after adolescent alcohol exposure. Alcoholism, Clinical and Experimental Research. https://doi.org/10.1111/acer.13599 (Original work published 2018)

Mulholland, P., & Prendergast, M. (2002). Post-insult exposure to (+/-) kavain potentiates N-methyl-D-aspartate toxicity in the developing hippocampus. Brain Research, 945(1), 106-13. https://doi.org/10.1016/s0006-8993(02)02745-2 (Original work published 2002)

Prendergast, M., Rogers, D., Mulholland, P., Littleton, J., LH, W. , Jr, Self, R., & Nath, A. (2002). Neurotoxic effects of the human immunodeficiency virus type-1 transcription factor Tat require function of a polyamine sensitive-site on the N-methyl-D-aspartate receptor. Brain Research, 954(2), 300-7. https://doi.org/10.1016/s0006-8993(02)03360-7 (Original work published 2002)

Mulholland, P., & Prendergast, M. (2003). Transection of intrinsic polysynaptic pathways reduces N-methyl-D-aspartate neurotoxicity in hippocampal slice cultures. Neuroscience Research, 46(3), 369-76. https://doi.org/10.1016/s0168-0102(03)00102-0

Mulholland, P., Harris, B., Wilkins, L., Self, R., Blanchard, J., Holley, R., … Prendergast, M. (1969). Opposing effects of ethanol and nicotine on hippocampal calbindin-D28k expression. Alcohol (Fayetteville, N.Y.), 31(1-2), 1-10. https://doi.org/10.1016/j.alcohol.2003.09.001 (Original work published 1969)

Self, R., Mulholland, P., Nath, A., Harris, B., & Prendergast, M. (2004). The human immunodeficiency virus type-1 transcription factor Tat produces elevations in intracellular Ca2+ that require function of an N-methyl-D-aspartate receptor polyamine-sensitive site. Brain Research, 995(1), 39-45. https://doi.org/10.1016/j.brainres.2003.09.052 (Original work published 2004)

Mulholland, P., Self, R., Harris, B., Littleton, J., & Prendergast, M. (-)-nicotine ameliorates corticosterone’s potentiation of N-methyl-d-aspartate receptor-mediated cornu ammonis 1 toxicity. Neuroscience, 125(3), 671-82. https://doi.org/10.1016/j.neuroscience.2004.02.007 (Original work published 2004)

Mulholland, P., Self, R., Harris, B., Littleton, J., & Prendergast, M. (2004). Choline exposure reduces potentiation of N-methyl-D-aspartate toxicity by corticosterone in the developing hippocampus. Brain Research. Developmental Brain Research, 153(2), 203-11. https://doi.org/10.1016/j.devbrainres.2004.08.008 (Original work published 2004)

Butler, T., Self, R., Smith, K., Sharrett-Field, L., Berry, J., Littleton, J., … Prendergast, M. (2010). Selective vulnerability of hippocampal cornu ammonis 1 pyramidal cells to excitotoxic insult is associated with the expression of polyamine-sensitive N-methyl-D-asparate-type glutamate receptors. Neuroscience, 165(2), 525-34. https://doi.org/10.1016/j.neuroscience.2009.10.018 (Original work published 2010)